, 2001). This constrained malignant development relates prominently towards the activation of p53 in aspiring cancer cells enduring telomere dysfunction. Inactivation of the p53 tumor suppressor is Probably the most Repeated functions in human epithelial cancers (Brosh and Rotter, 2009) and the position of p53 can dictate no matter whether https://laneifmvz.qodsblog.com/28359908/detailed-notes-on-sanakin-tedavisi-yan-etkileri